CD33 splicing SNP regulates expression levels of CD33 in normal regenerating monocytes in AML patients.

Creator(s)

Jatinder K. Lamba
Andrew P. Voigt
Lata Chauhan
Miyoung Shin
Richard Aplenc
Lisa Eidenschink Brodersen
Alan S. Gamis, Children's Mercy HospitalFollow
Soheil Meshinchi
Michael R. Loken

Document Type

Letter to the Editor

Publication Date

9-2018

Identifier

DOI: 10.1080/10428194.2017.1421756; PMCID: PMC7199442

Journal Title

Leukemia & lymphoma

Volume

59

Issue

9

First Page

2250

Last Page

2253

MeSH Keywords

Acute Disease; Alleles; Alternative Splicing; Child; Gene Expression Regulation; Gene Frequency; Genotype; HEK293 Cells; Humans; K562 Cells; Leukemia, Myeloid; Monocytes; Polymorphism, Single Nucleotide; Sialic Acid Binding Ig-like Lectin 3

Keywords

Acute Disease; Alleles; Alternative Splicing; Child; Gene Expression Regulation; Gene Frequency; Genotype; HEK293 Cells; Humans; K562 Cells; Leukemia, Myeloid; Monocytes; Polymorphism, Single Nucleotide; Sialic Acid Binding Ig-like Lectin 3

Recommended Citation

Lamba JK, Voigt AP, Chauhan L, et al. CD33 splicing SNP regulates expression levels of CD33 in normal regenerating monocytes in AML patients. Leuk Lymphoma. 2018;59(9):2250-2253. doi:10.1080/10428194.2017.1421756